Participating Institutes
Ludwig Boltzmann Institute for Cardiosurgical Research
Department of Cardiothoracic Surgery, Institute for Biomedical Research and Institute for Biomedical Engineering, all General Hospital Vienna, Vienna, Austria
Ludwig Boltzmann Institute for Cardiovascular Research
Department of Internal Medicine II, General Hospital Vienna, Vienna, Austria
Ludwig Boltzmann Institute for Arrhythmia Research
Department of Internal Medicine III, Wilhelminenhospital, Vienna, Austria
Goal of the cluster
Cardiovascular diseases have been the leading cause of death in Europe during the last decades and will remain in this position also in the near future in spite of changes in life style. The reasons are on the one hand an ageing population, on the other hand paradoxically the advances in cardiology and cardiovascular surgery with improved survival after acute myocardial infarction but consecutive development of heart failure in the long run.
The three institutions, mentioned above, have worked during the last decades fighting heart diseases in the fields of cardiac surgery and cardiology separately. The future of research however lies in an integrated and interdisciplinary approach including basic and clinical science without the classic separations in surgery and internal medicine. It is the goal of the Ludwig Boltzmann Cluster (LBC) for Cardiovascular Research to act in this integrated and interdisciplinary manner and to address common problems in cardiovascular research focussing on pathophysiology, diagnosis and therapy.
Office
Fr. Eva Klausner
Währinger Gürtel 18-20, AKH, Leitstelle 1Q
1090 Vienna
Austria / European Union
| P: +43 1 40400 5221 | F: +43 1 40400 5229 |
| E: eva.klausner(0)meduniwien.ac.at | W: center-biomed-research |
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Recent Publications
- Optimizing reperfusion therapy in acute ST-elevation myocardial infarction by a pharmaco-invasive treatment approach in a well-organized network.
- Oncostatin M and IL-6 induce u-PA and VEGF in prostate cancer cells and correlate in vivo.
- Antiplatelet function variability in clopidogrel-treated patients: need for new antiplatelet agents.
- Interleukin-33 induces expression of adhesion molecules and inflammatory activation in human endothelial cells and in human atherosclerotic plaques.
- Complementary role of copeptin and high-sensitivity troponin in predicting outcome in patients with stable chronic heart failure.

