Institute of Immunology
Medical University of Vienna
Borschkegasse 8a
Tel: +43-1-4277-64951
Fax: +43-1-1-4277-9649
Email: johannes.stoeckl@meduniwien.ac.at
Website: http://www.meduniwien.ac.at/immunologie
Research interests
The major research interest of my group is to unravel the immune evasion mechanisms used by human rhinoviruses (HRV) in order to identify molecular checkpoints of the immune system and to better understand the pathogenesis of the common cold, one of the most frequent infectious diseases in humans.
Selected publications
Kirchberger, S., O. Majdic, P. Steinberger, S. Bluml, K. Pfistershammer, G. Zlabinger, L. Deszcz, E. Kuechler, W. Knapp, and J. Stockl. 2005. Human rhinoviruses inhibit the accessory function of dendritic cells by inducing sialoadhesin and B7-H1 expression. J Immunol 175:1145.
Bluml, S., G. Zupkovitz, S. Kirchberger, M. Seyerl, V. N. Bochkov, K. Stuhlmeier, O. Majdic, G. J. Zlabinger, C. Seiser, and J. Stockl. 2009. Epigenetic regulation of dendritic cell differentiation and function by oxidized phospholipids. Blood 114:5481.
Schrauf, C., S. Kirchberger, O. Majdic, M. Seyerl, G. J. Zlabinger, K. M. Stuhlmeier, M. Sachet, J. Seipelt, and J. Stockl. 2009. The ssRNA genome of human rhinovirus induces a type I IFN response but fails to induce maturation in human monocyte-derived dendritic cells. J Immunol 183:4440.
Seyerl, M., S. Kirchberger, O. Majdic, J. Seipelt, C. Jindra, C. Schrauf, and J. Stockl. 2009. Human rhinoviruses induce IL-35-producing Treg via induction of B7-H1 (CD274) and sialoadhesin (CD169) on DC. Eur J Immunol 40:321.
