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Philipp Hohensinner
Dr. Philipp Hohensinner

Center for Biomedical Research (Division of Biomedical Research)
Position: Research Associate (Postdoc)

ORCID: 0000-0003-4819-3190
T +43 1 40400 73515


Aging, Premature; Atherosclerosis; Macrophages; Monocytes

Research group(s)

Research interests

My current main research interest is to analyze the function and functional changes of the innate immune system in health and disease with a special focus on cardiovascular disease.

Techniques, methods & infrastructure

  • In vitro cell culture,
  • animal models,
  • qPCR,
  • flow cytometry,
  • molecular biology techniques

Selected publications

  1. Haider, P. et al. (2020) ‘Neutrophil Extracellular Trap Degradation by Differently Polarized Macrophage Subsets’, Arteriosclerosis, Thrombosis, and Vascular Biology, 40(9), pp. 2265–2278. Available at:
  2. Hohensinner, P. et al. (2021) 'Alternative activation of human macrophages enhances tissue factor expression and production of extracellular vesicles', Haematologica 2021 available at 10.3324/haematol.2019.220210
  3. Thaler, B. et al. (2019) ‘Differential expression of Plg-RKT and its effects on migration of proinflammatory monocyte and macrophage subsets’, Blood, 134(6), pp. 561–567. Available at:
  4. Hohensinner, P.J. et al. (2017) ‘PAI-1 (Plasminogen Activator Inhibitor-1) Expression Renders Alternatively Activated Human Macrophages Proteolytically Quiescent’, Arteriosclerosis, Thrombosis, and Vascular Biology, 37(10), pp. 1913–1922. Available at:
  5. Li, Y. et al. (2016) ‘Deficient Activity of the Nuclease MRE11A Induces T Cell Aging and Promotes Arthritogenic Effector Functions in Patients with Rheumatoid Arthritis’, Immunity, 45(4), pp. 903–916. Available at: