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Philipp Hohensinner
Dr. Philipp Hohensinner

Department of Medicine II (Division of Cardiology)
Position: Research Associate (Postdoc)

ORCID: 0000-0003-4819-3190
T +43 1 40400 73515


Aging, Premature; Atherosclerosis; Macrophages; Monocytes

Research interests

My current main research interest is to define cellular changes within an atherosclerotic plaque.

Techniques, methods & infrastructure

  • In vitro cell culture,
  • animal models,
  • qPCR,
  • flow cytometry,
  • molecular biology techniques

Selected publications

  1. Thaler, B. et al., 2016. Differential in vivo activation of monocyte subsets during low-grade inflammation through experimental endotoxemia in humans. Scientific Reports, 6(1). Available at:
  2. Hohensinner, P.J. et al., 2016. Reduced Ang2 expression in aging endothelial cells. Biochemical and Biophysical Research Communications, 474(3), pp.447-451. Available at:
  3. Hohensinner, P.J. et al., 2016. Age intrinsic loss of telomere protection via TRF1 reduction in endothelial cells. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1863(2), pp.360-367. Available at:
  4. Li, Y. et al., 2016. Deficient Activity of the Nuclease MRE11A Induces T Cell Aging and Promotes Arthritogenic Effector Functions in Patients with Rheumatoid Arthritis. Immunity, 45(4), pp.903-916. Available at:
  5. Hohensinner, P.J., Goronzy, J.J. & Weyand, C.M., 2014. Targets of Immune Regeneration in Rheumatoid Arthritis. Mayo Clinic Proceedings, 89(4), pp.563-575. Available at: